What is a parietal occipital subarachnoid hemorrhage?
Question:
Answers:
parietal= bone that forms the roof and sides of skull
occipial=bone that forms the base of the skull
subarachnoid=under the weblike middle membrane of meninges
hemorrhage=bleeding inside
basically bleeding in your brain, and yes it could probably be from the vacuum used during delivery of the baby, their heads are the most fragile.
Other Answers:
All I know is that you just named parts of the skull.sorry thats all I know
It is a bleed in the sides and front deep in the brain and I do think a Mighty Vac could cause this if used improperly. Intracranial Hemorrhage
Last Updated: April 18, 2006
Synonyms and related keywords: intracerebral hemorrhage, intraparenchymal hemorrhage, intracranial hematoma, intracerebral hematoma, intraparenchymal hematoma, epidural hematoma, subdural hematoma, subarachnoid hemorrhage, intraventricular hemorrhage
AUTHOR INFORMATION Section 1 of 11 Cli
Author: David S Liebeskind, MD, Neurology Director, Stroke Imaging; Associate Neurology Director, UCLA S, Assistant Professor of Neurology, Department of Neurology, University of California at Los Angeles
David S Liebeskind, MD, is a member of the following medical societies: American Academy of Neurology, American Heart Association, American Medical Association, American Society of Neuroimaging, American Society of Neuroradiology, National Stroke Association, and Stroke Council of the American Heart Association
Editor(s): Jeffrey L Saver, MD, Head of Stroke Neurology and Director of Stroke Unit, Professor, Department of Neurology, University of California at Los Angeles Medical Center; Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine; Howard Kirshner, MD, Vice-Chair, Professor, Department of Neurology, Vanderbilt University School of Medicine; Selim R Benbadis, MD, Professor, Director of Comprehensive Epilepsy Program, Departments of Neurology and Neurosurgery, University of South Florida College of Medicine; and Helmi L Lutsep, MD, Associate Director, Oregon Stroke Center; Associate Professor, Department of Neurology, Oregon Health and Science University
Disclosure
INTRODUCTION Section 2 of 11 Click here to go to the previous section in this topic Click here to go to the top of this page Click here to go to the next section in this topic
Author Information Introduction Clinical Differentials Workup Treatment Medication Follow-up Miscellaneous Pictures Bibliography
Background: Intracranial hemorrhage (ie, the pathological accumulation of blood within the cranial vault) may occur within brain parenchyma or the surrounding meningeal spaces. Hemorrhage within the meninges or the associated potential spaces, including epidural hematoma, subdural hematoma, and subarachnoid hemorrhage, is covered in detail in other articles. Intracerebral hemorrhage (ICH) and extension of parenchymal bleeding into the ventricles (ie, intraventricular hemorrhage [IVH]) are detailed here. ICH accounts for 8-13% of all strokes and results from a wide spectrum of disorders. ICH is more likely to result in death or major disability than ischemic stroke or subarachnoid hemorrhage. ICH and accompanying edema may disrupt or compress adjacent brain tissue, leading to neurological dysfunction. Substantial displacement of brain parenchyma may cause elevation of intracranial pressure (ICP) and potentially fatal herniation syndromes.
Pathophysiology: Nontraumatic ICH most commonly results from hypertensive damage to blood vessel walls (eg, hypertension, eclampsia, drug abuse), but it also may be due to autoregulatory dysfunction with excessive cerebral blood flow (eg, reperfusion injury, hemorrhagic transformation, cold exposure), rupture of an aneurysm or arteriovenous malformation (AVM), arteriopathy (eg, cerebral amyloid angiopathy, moyamoya), altered hemostasis (eg, thrombolysis, anticoagulation, bleeding diathesis), hemorrhagic necrosis (eg, tumor, infection), or venous outflow obstruction (eg, cerebral venous thrombosis). Nonpenetrating and penetrating cranial trauma are also common causes of ICH.
Chronic hypertension produces a small vessel vasculopathy characterized by lipohyalinosis, fibrinoid necrosis, and development of Charcot-Bouchard aneurysms, affecting penetrating arteries throughout the brain including lenticulostriates, thalamoperforators, paramedian branches of the basilar artery, superior cerebellar arteries, and anterior inferior cerebellar arteries.
Predilection sites for ICH include the basal ganglia (40-50%), lobar regions (20-50%), thalamus (10-15%), pons (5-12%), cerebellum (5-10%), and other brainstem sites (1-5%).
IVH occurs in one third of ICH cases from extension of thalamic ganglionic bleeding into the ventricular space. Isolated IVH frequently arise from subependymal structures including the germinal matrix, AVMs, and cavernous angiomas.
Frequency:
* In the US: Each year, ICH affects approximately 12-15 per 100,000 individuals, including 350 hypertensive hemorrhages per 100,000 elderly individuals. The overall incidence of ICH has declined since the 1950s.
* Internationally: Asian countries have a higher incidence of ICH than other regions of the world.
Mortality/Morbidity:
* Annually, more than 20,000 individuals in the United States die of ICH.
* ICH has a 30-day mortality rate of 44%.
* Pontine or other brainstem ICH has a mortality rate of 75% at 24 hours.
Race: ICH has a higher incidence among populations with a higher frequency of hypertension, including African Americans. A higher incidence of ICH has been noted in Chinese, Japanese, and other Asian populations, possibly due to environmental factors (eg, a diet rich in fish oils) and/or genetic factors.
Sex: ICH has a slight male predominance, though study results have been conflicting.
* Cerebral amyloid angiopathy may be more common among women.
* Phenylpropanolamine use has been associated with ICH in young women.
Age: Incidence of ICH increases in individuals older than 55 years and doubles with each decade until age 80 years.
* The relative risk of ICH is greater than 7 in individuals older than 70 years.
* In individuals younger than 45 years, lobar hemorrhage is the most common site of and frequently is associated with AVMs.
* Subependymal hemorrhage or germinal matrix hemorrhage is primarily seen in premature infants.
CLINICAL Section 3 of 11 Click here to go to the previous section in this topic Click here to go to the top of this page Click here to go to the next section in this topic
Author Information Introduction Clinical Differentials Workup Treatment Medication Follow-up Miscellaneous Pictures Bibliography
History:
* Onset of symptoms of ICH is usually during daytime activity, with progressive (ie, minutes to hours) development of the following:
o Alteration in level of consciousness (approximately 50%)
o Nausea and vomiting (approximately 40-50%)
o Headache (approximately 40%)
o Seizures (approximately 6-7%)
o Focal neurological deficits
* Lobar hemorrhage due to cerebral amyloid angiopathy may be preceded by prodromal symptoms of focal numbness, tingling, or weakness.
* A history of hypertension, trauma, illicit drug abuse, or a bleeding diathesis may be elicited.
Physical: Clinical manifestations of ICH are determined by the size and location of hemorrhage, but may include the following:
* Hypertension, fever, or cardiac arrhythmias
* Nuchal rigidity
* Subhyaloid retinal hemorrhages
* Altered level of consciousness
* Anisocoria
* Focal neurological deficits
o Putamen - Contralateral hemiparesis, contralateral sensory loss, contralateral conjugate gaze paresis, homonymous hemianopia, aphasia, neglect, or apraxia
o Thalamus - Contralateral sensory loss, contralateral hemiparesis, gaze paresis, homonymous hemianopia, miosis, aphasia, or confusion
o Lobar - Contralateral hemiparesis or sensory loss, contralateral conjugate gaze paresis, homonymous hemianopia, abulia, aphasia, neglect, or apraxia
o Caudate nucleus - Contralateral hemiparesis, contralateral conjugate gaze paresis, or confusion
o Brain stem - Quadriparesis, facial weakness, decreased level of consciousness, gaze paresis, ocular bobbing, miosis, or autonomic instability
o Cerebellum - Ataxia, usually beginning in the trunk, ipsilateral facial weakness, ipsilateral sensory loss, gaze paresis, skew deviation, miosis, or decreased level of consciousness
Causes:
* Hypertension
* Arteriovenous malformation
* Aneurysmal rupture
* Cerebral amyloid angiopathy
* Intracranial neoplasm
* Coagulopathy
* Hemorrhagic transformation of an ischemic infarct
* Cerebral venous thrombosis
* Sympathomimetic drug abuse
* Moyamoya
* Sickle cell disease
* Eclampsia or postpartum vasculopathy
* Infection
* Vasculitis
* Neonatal IVH
* Trauma
Contact a Pedatric Neurologist for possible causation of this problem as soon as possible. They can assess the likelihood of delivery trauma.
http://search.yahoo.com/search?search=parietal+occipital+subarachnoid+hemorrhage&ei=UTF-8&fr=ks-ques&p=parietal+occipital+subarachnoid+hemorrhage
More Questions and Answers
- i'm gasping for ciggy, but dr told me to give up. tell me joke, take mind off it?
- I think I have broken at least 1 rib, but apparently doctors cant really help. Is it worth me going to see?
- can i re-hydrate by drinking decaffeinated coffee?
- How many times did Joshua nd his people march around jericho before the walls fell down?
- How do you heal a bruised shoulder muscle asap?
- My 7 yr old was served raw chicken and accidentally took 2 bites.what do I watch for?? Or do??
- What is the best way to stop a bloody nose?
- pain from sprained ankle?